Spatiotemporal AMPKα2 deletion in mice induces cardiac dysfunction, fibrosis and cardiolipin remodeling associated with mitochondrial dysfunction in males only.

"Declarations Ethics approval and consent to participateNot applicable. Consent for publicationNot applicable. Competing interestsNo potential conflicts of interest were disclosed. Competing interests No potential conflicts of interest were disclosed."

"Funding Our laboratory is a member of the Laboratory of Excellence LERMIT and is supported by grants from “Fondation pour la Recherche Médicale” (to A.G., #DPM20121125546), European Research Area Network on Cardiovascular Diseases (to J.P, #ANR-19-ECVD-0007-01), Fondation de France (to A.G., 00086500), Université Paris-Sud (ERM) and CORDDIM (to R.V.-C, #cod110153), STEFANIK (to C. L. and M. N., 35814NK). This study was supported by the Slovak Research and Development Agency grant (M.N., APVV-15-0302), and by the Slovak Scientific Grant Agency (M.N., VEGA 2/0091/19). R.V.-C is emeritus scientist at CNRS."