Carboxyl truncation of α-synuclein occurs early and is influenced by human APOE genotype in transgenic mouse models of α-synuclein pathogenesis.

"Declarations Ethics approval and consent to participateAll procedures were performed according to the National Institute of Health Guide for the Care and Use of Experimental Animals and were approved by the University of Florida Institutional Animal Care and Use Committee. Competing interestsWe declare no competing interest in this manuscript. Competing interests We declare no competing interest in this manuscript."

"This work was supported by grants from the National Institute of Neurological Disorders and Stroke (R01NS089022, R01NS100876, RF1NS129567, RF1AG057933). G.M.L. was supported by T32NS082168 training grant from National Institute of Neurological Disorders and Stroke. Z.A.S. was supported by fellowship F30AG063446 from the National Institute on Aging."