Circ_072697 knockdown promotes advanced glycation end products-induced cell proliferation and migration in HaCaT cells via miR-3150a-3p/KDM2A axis.

Journal Information

Full Title: BMC Endocr Disord

Abbreviation: BMC Endocr Disord

Country: Unknown

Publisher: Unknown

Language: N/A

Publication Details

Subject Category: Endocrinology

Available in Europe PMC: Yes

Available in PMC: Yes

PDF Available: No

Transparency Score
3/6
50.0% Transparent
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Evidence found in paper:

"Declarations Competing interestsThe authors declare that they have no competing interests. Ethics approval and consent to participateAll processes were performed based on the Declaration of Helsinki and received ethics approval from ethics committee of Ruijin Hospital, Shanghai Jiaotong University School of Medicine (2019-85). The written informed consents were obtained from participants prior to the start of this research. Consent for publicationNot applicable. Competing interests The authors declare that they have no competing interests."

Evidence found in paper:

"Funding This study is supported by the National Natural Science Foundation of China (No. 81401163 and No. 81670752), and Construction Project of Special Disease in Putuo District -- Arteriosclerosis Obliterans of Lower Extremity (No. 2020tszb01)."

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Open Access
Paper is freely available to read
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Tool: rtransparent

OST Version: N/A

Last Updated: Aug 05, 2025