Circ_072697 knockdown promotes advanced glycation end products-induced cell proliferation and migration in HaCaT cells via miR-3150a-3p/KDM2A axis.
Journal Information
Full Title: BMC Endocr Disord
Abbreviation: BMC Endocr Disord
Country: Unknown
Publisher: Unknown
Language: N/A
Publication Details
Subject Category: Endocrinology
Available in Europe PMC: Yes
Available in PMC: Yes
PDF Available: No
Related Papers from Same Journal
Transparency Score
Transparency Indicators
Click on green indicators to view evidence textCore Indicators
"Declarations Competing interestsThe authors declare that they have no competing interests. Ethics approval and consent to participateAll processes were performed based on the Declaration of Helsinki and received ethics approval from ethics committee of Ruijin Hospital, Shanghai Jiaotong University School of Medicine (2019-85). The written informed consents were obtained from participants prior to the start of this research. Consent for publicationNot applicable. Competing interests The authors declare that they have no competing interests."
"Funding This study is supported by the National Natural Science Foundation of China (No. 81401163 and No. 81670752), and Construction Project of Special Disease in Putuo District -- Arteriosclerosis Obliterans of Lower Extremity (No. 2020tszb01)."
Additional Indicators
Assessment Info
Tool: rtransparent
OST Version: N/A
Last Updated: Aug 05, 2025