Acidosis-mediated increase in IFN-γ-induced PD-L1 expression on cancer cells as an immune escape mechanism in solid tumors.

"Declarations Ethics approval and consent to participateStudy approvalAll animal experiments were approved by the Regierungspräsidium Tübingen and were in concordance with German federal regulations. The veterinarians at the University of Tübingen were consulted for conducting in vivo animal experiments. Consent for publicationNot applicable. Competing interestsThe authors declare no competing interests. Competing interests The authors declare no competing interests."

"Funding Open Access funding enabled and organized by Projekt DEAL. This work was supported by the Cluster of Excellence iFIT (EXC 2180) “Image Guided and Functionally Instructed Tumor Therapies”, University of Tuebingen, Germany, Wilhelm Sander Foundation (2020.100.1), and the Werner Siemens Foundation."